Anti-Aging Research

Understanding aging as a biological process — and how metabolism, genetics, inflammation, and cellular repair influence longevity.

Core Anti-Aging Domains

Aging is driven by cellular damage, telomere shortening, mitochondrial dysfunction, and loss of repair mechanisms.

Focus: cellular stress, oxidative damage, and regeneration limits.

Long-term low-grade inflammation accelerates aging and increases risk of metabolic and neurodegenerative disorders.

Inflammaging is a core driver of biological decline.

Insulin sensitivity, glucose regulation, and lipid metabolism strongly influence lifespan and healthspan.

Diet, fasting, and activity directly impact aging speed.

VO₂ max reflects the body’s oxygen utilization capacity and is one of the strongest predictors of longevity.

Regular aerobic activity can slow age-related decline.

HRV measures nervous system resilience and stress adaptability. Higher HRV is associated with better recovery and longevity.

Breathing, sleep, and recovery are key regulators.

Genes influence aging, but epigenetic changes determine how genes are expressed over time.

Lifestyle can slow or accelerate epigenetic aging clocks.

Cell reprogramming, senolytics, gene therapy, AI-driven biomarkers, and biological robotics may redefine aging in the coming decades.

Experimental today — foundational tomorrow.